STDs MY FRIEND. BE CAREFUL

 

1. Picture of Genital Warts (HPV)

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Genital wart: A wart in the moist skin of the genitals or around the anus. Genital warts are due to a human papillomavirus (HPV). The HPVs, including those that cause genital warts, are transmitted through sexual contact. HPV can also be transmitted from mother to baby during childbirth. Most people infected with HPV have no symptoms, but these viruses increase a woman's risk for cancer of the cervix. HPV infection is the most common sexually transmitted disease in the US. It is also the leading cause of abnormal PAP smears and pre-cancerous changes of the cervix in women. There is no cure for HPV infection, although anti-viral medications can reduce outbreaks and topical preparations can speed healing. Once contracted, the virus can stay with a person for life. Also called condyloma acuminatum, condylomata.

 

2. Picture of Crabs (Pubic Lice)

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Crabs: Slang for pubic lice, parasitic insects that can infest in the genital area of humans. Pubic lice are usually spread through sexual contact. Rarely, infestation can be spread through contact with an infested person's bed linens, towels, or clothes. The key symptom of pubic lice is itching in the genital area. Lice eggs (nits) or crawling lice can be seen with the naked eye.

3. Picture of The Clap (Gonorrhea)

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Gonorrhea: A bacterial infection that is transmitted by sexual contact. Gonorrhea is one of the oldest known sexually transmitted diseases (STDs), and it is caused by the Neisseria gonorrhoeae bacteria. Men with gonorrhea may have a yellowish discharge from the penis accompanied by itching and burning. More than half of women with gonorrhea do not have any symptoms. If symptoms occur, they may include burning or frequent urination, yellowish vaginal discharge, redness and swelling of the genitals, and a burning or itching of the vaginal area. If untreated, gonorrhea can lead to severe pelvic infections and even sterility. Complications in later life can include inflammation of the heart valves, arthritis, and eye infections. Gonorrhea can also cause eye infections in babies born of infected mothers. Gonorrhea is treated with antibiotics.

4. Picture of Syphilis

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Syphilis: A sexually transmitted disease caused by Treponema pallidum, a microscopic organism called a spirochete. This worm-like, spiral-shaped organism infects people by burrowing into the moist mucous membranes of the mouth or genitals. From there, the spirochete produces a non-painful ulcer known as a chancre. There are three stages of syphilis:

• The first (primary) stage: This involves the formation of the chancre. At this stage, syphilis is highly contagious. The primary stage can last one to five weeks. The disease can be transmitted from any contact with one of the ulcers, which are teeming with spirochetes. If the ulcer is outside of the vagina or on the scrotum, the use of condoms may not help in preventing transmission. Likewise, if the ulcer is in the mouth, merely kissing the infected individual can spread syphilis. Even without treatment, the early infection resolves on its own in most women.

• The second (secondary) stage: However, 25 percent of cases will proceed to the secondary stage of syphilis, which lasts four to six weeks. This phase can include hair loss; a sore throat; white patches in the nose, mouth, and vagina; fever; headaches; and a skin rash. There can be lesions on the genitals that look like genital warts, but are caused by spirochetes rather than the wart virus. These wart-like lesions, as well as the skin rash, are highly contagious. The rash can occur on the palms of the hands, and the infection can be transmitted by casual contact.

• The third (tertiary) stage: This final stage of the disease involves the brain and heart, and is usually no longer contagious. At this point, however, the infection can cause extensive damage to the internal organs and the brain, and can lead to death.

Diagnosis is by blood test, either the rapid plasma reagin (RPR) or Venereal Disease Research Laboratory (VDRL) test. Treatment is with antibiotics.
Syphilis remains a major health problem. About 12 million new cases of syphilis occur every year. More than 90% of them are in developing nations where congenital syphilis remains a leading cause of stillbirths and newborn deaths. In North America and Western Europe, syphilis is disproportionately common and rising among men who have sex with men and among persons who use cocaine or other illicit drugs.
The name "syphilis" was coined by Hieronymus Fracastorius (Girolamo Fracastoro). Fracastorius was a true Renaissance man; he wrote on the temperature of wines, the rise of the Nile, poetry, the mind, and the soul; he was an astronomer, geographer, botanist, mathematician, philosopher and, last but not least in the present context, a physician. In 1530 he published the poem "Syphilis sive morbus gallicus" (Syphilis or the French Disease) in which the name of the disease first appeared. Perhaps more importantly, Fracastorius went on in 1546 to write "On Contagion" ("De contagione et contagiosis morbis et curatione"), the first known discussion of the phenomenon of contagious infection: a landmark in the history of infectious disease.

 

5. Picture of Chlamydia

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Chlamydia: The agent of a sexually transmitted disease, a type of bacteria found in the cervix, urethra, throat, or rectum that acts very much like gonorrhea in the way it is spread, the symptoms it produces, and its long-term consequences. Chlamydia is destructive to the Fallopian tubes, causing infertility, tubal pregnancy, and severe pelvic infection. It is common for infected women to have no symptoms. Chlamydia is associated with an increased incidence of preterm births. Also, an infant can acquire the disease during passage through the birth canal, leading to eye problems or pneumonia. Chlamydia is one of the reasons newborns are routinely treated with antibiotic eyedrops. Chlamydia can also cause inflammation of the urethra, epididymis, and rectum in men. A chronic form of arthritis, called reactive arthritis, can develop after chlamydia infection.

6. Picture of Herpes Simplex Virus Type 1

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Herpes simplex type 1: A herpes virus that causes cold sores and fever blisters in and around the mouth. Here is a depiction of a typical fever blister caused by herpes simplex 1:
In rare cases, as when someone's immune system is severely compromised, this virus can cause infection of the brain (encephalitis), requiring intravenous therapy.
Herpes simplex type 1 is also known as human herpesvirus 1 (HHV-1).

7. Picture of Herpes Simplex Virus Type 2

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Herpes simplex type 2: A herpes virus that causes genital herpes, which is characterized by sores in the genital area. Genital herpes is a sexually transmitted disease (STD).
This virus, like herpes simplex type 1, can also cause infection of the brain (encephalitis) if the immune system is severely defective or compromised. The treatment of infection with herpes simplex type 2 is by topical or oral anti-viral medication.
Herpes simplex type 2 is also known as human herpesvirus 2 (HHV-2).

8. Picture of Hepatitis B

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Hepatitis B: Inflammation of the liver due to the hepatitis B virus (HBV), once thought to be passed only through blood products. It is now known that hepatitis B can also be transmitted via needle sticks, body piercing and tattooing using un sterilized instruments, the dialysis process, sexual and even less intimate close contact, and childbirth. Symptoms include fatigue, jaundice, nausea, vomiting, dark urine, light stools. Diagnosis is by blood test. Treatment is via anti-viral drugs and/or hepatitis B immunoglobulin (HBIG).
Chronic hepatitis B may be treated with a variety of medications. Healthcare workers accidentally exposed to materials infected with hepatitis B and individuals with known sexual contact with hepatitis B patients are usually given both HBIG and the hepatitis B vaccine to provide both immediate and long-term protection. HBV infection can be prevented by the hepatitis B vaccine, and by avoiding activities that could lead to getting the virus. Hepatitis B was formerly referred to as serum hepatitis.

9. Picture of HIV/AIDS

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HIV: Acronym for the Human Immunodeficiency Virus, the cause of AIDS (acquired immunodeficiency syndrome). HIV has also been called the human lymphotropic virus type III, the lymphadenopathy-associated virus and the lymphadenopathy virus. No matter what name is applied, it is a retrovirus. (A retrovirus has an RNA genome and a reverse transcriptase enzyme. Using the reverse transcriptase, the virus uses its RNA as a template for making complementary DNA which can integrate into the DNA of the host organism).
Although the American research Robert Gallo at the National Institutes of Health (NIH) believed he was the first to find HIV, it is now generally accepted that the French physician Luc Montagnier (1932-) and his team at the Pasteur Institute discovered HIV in 1983-84

10. Picture of Trichomoniasis

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Trichomoniasis: Infection with trichomonas, in humans with Trichomonas vaginalis.

11. Picture of Chancroid

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Chancroid: A sexually transmitted infection (STI) caused by the bacterium Haemophilus ducreyi. Periodic outbreaks of chancroid have occurred in the US, usually in minority populations in the inner cities. This disease is common in sub-Saharan Africa among men who have frequent contact with prostitutes.
The infection begins with the appearance of painful open sores on the genitals, sometimes accompanied by swollen, tender lymph nodes in the groin. These symptoms occur within a week after exposure. Symptoms in women are often less noticeable and may be limited to painful urination or defecation, painful intercourse, rectal bleeding, or vaginal discharge.
Chancroid can be treated effectively with several antibiotics. Chancroid is one of the genital ulcer diseases associated with an increased risk of transmission of the human immunodeficiency virus (HIV), the cause of AIDS.
Also known as soft chancre, soft sore, and soft ulcer.

12. Picture of LGV (Lymphogranuloma Venereum)

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Lymphogranuloma venereum: Abbreviated LGV. An uncommon genital or anorectal (affecting the anus and/or rectum) infection that is caused by a specific type of Chlamydia trachomatis.
Patients typically have tender glands (lymph nodes) in the groin and may recently have had a genital ulcer that resolved on its own. Other patients, in particular those with HIV infection, may have rectal or anal inflammation, scarring, and narrowing (stricture), which cause frequent small bowel movements (diarrhea) and a sense of incomplete evacuation of the bowels. In addition, these patients can have pain around the anal area (perianal), and occasionally drainage from the perianal area or the glands in the groin.
The diagnosis of lymphogranuloma venereum is suspected in a person with typical symptoms and in whom other causes (such as chancroid, herpes, and syphilis) have been excluded. The diagnosis in such a patient is usually made by a blood test that detects specific antibodies to Chlamydia, which are produced as part of the body's immunological (defensive) response to that bacterial organism.
Once lymphogranuloma venereum is diagnosed, it is usually treated with doxycycline 100 mg twice per day by mouth for 21 days. If this is not an option, for example, because of intolerance to the drug, azithromycin 2g in one dose can be used as an alternative.
A person who has been sexually exposed to a person with lymphogranuloma venereum should be examined for signs or symptoms of lymphogranuloma venereum, as well as for chlamydial infection of the urethra, since the lymphogranuloma venereum and urethral strains (types) of Chlamydia trachomatis can co-exist. If exposure occurred within 30 days of the onset of their partner's symptoms of lymphogranuloma venereum, the exposed person should be treated.

 

you need to know about HPV.

What Is HPV?

HPV stands for human papillomavirus. It's a virus that can be transmitted through sexual contact. During intercourse or oral sex, HPV can make its way into the genitals, mouth, or throat and cause infection.
Sexually transmitted HPV comes in more than 40 different varieties. The type of the virus you get determines what effects it has on your body. Certain types of HPV cause genital warts. Other HPV types can make cells turn cancerous. You've probably heard that HPV causes cervical cancer, but it also causes less common cancers of the vulva, vagina, penis, anus, head, and neck.
What's tricky about HPV is that it doesn't have symptoms. There's no sore throat or fever to let you know you've been infected. Most people clear the infection on their own. In fact, you might have absolutely no idea you've been infected until you develop genital warts or have an abnormal Pap test.
Though HPV might not be as well known among sexually transmitted infections (STIs) as herpes or syphilis, it is actually the most common STI. If you're sexually active, there's a very good chance of being infected with HPV at some point in your life. That's why immunization is so important.

There are three HPV vaccines; what's the difference?

Three vaccines are available to protect against the types of HPV that cause most cervical, vaginal, vulvar, penile and anal cancers. They are Cervarix, Gardasil, and Gardasil-9. Gardasil, and Gardasil-9 also protect against most genital warts. Whichever of these vaccines you and your doctor choose, you should stick with the same vaccine for all three shots.

Do I have to get the HPV vaccine?

Whether you have to get vaccinated depends on your age and where you live. In certain states, girls and boys within the recommended ages may need to get the HPV vaccine in order to go to school.
Getting vaccinated could help lead to the reduction of the prevalence of HPV infection.  HPV is known to cause cervical, vaginal, vulvar, penile, and anal cancer as well ascancer of the back of the throat. Cervical cancer alone kills about 4,000 women each year in the U.S. One in four persons in the United States is infected with HPV and most of them are unaware. It spreads easily among infected partners.
The vaccine Cervarix protects against HPV-16 and HPV-18, which is responsible for 70% of all cervical cancers. Gardasil prevents these strains of HPV as well as HPV- 6 and HPV-11, which are known to cause 90% of all genital warts. Gardasil-9 also protects against these four HPV strains as well as five others.

When should I get the HPV immunization?

The best time to get the HPV vaccine is before you've started having sexual activity. That's why the CDC recommends that both boys and girls get their vaccination at age 11 or 12, although they can get the vaccine as early as age 9. If you're 13 or older and you haven't already been vaccinated, you can still get the vaccine through age 26.

If I'm over age 26, can I still get vaccinated?

The HPV vaccine isn't recommended for people over age 26, because it hasn't been studied well enough in this age group. If enough future studies show that it is safe and effective for people over 26, the FDA may eventually start recommending it for this age group.

How many shots do I need?

You'll get three shots of the HPV vaccine over a 6-month period. You need to take all three doses to be completely protected. You'll get the second shot about 1 to 2 months after the first, and the third shot 6 months after the first. Once you've started with a vaccine brand (Cervarix, Gardasil or Gardasil-9), stick with it for all three shots.

f I already have HPV, will this vaccine treat it?

No. If you have a current HPV, the vaccine won't get rid of the infection. However, if you have one type of HPV, the vaccine may prevent you from getting another type of the virus. There's really no way to treat the virus once you have it, although there are treatments for diseases caused by HPV such as genital warts and genital cancers. This is why you should have regular pelvic exams and Pap tests (if you're female) to screen for cervical cancer.

Does the HPV vaccine protect me for life?

The vaccine appears to offer long-term protection from HPV. However, even women who have received the vaccine should see their gynecologist regularly for a Pap test to check for cervical cancer, because the vaccine doesn’t protect against all HPV types that can cause cervical cancer.

Will my insurance cover the cost of the HPV vaccine?

Most insurance plans cover routine vaccines, which means that if you're in the recommended age group, your insurance should pay for the vaccine. Check with your insurance company just to be sure. If your family doesn't have health insurance or you're on Medicaid, you should be able to get the HPV vaccine for free through the Vaccines for Children (VFC) program.

Is this vaccination safe?

Vaccines have to be rigorously tested before they can be widely distributed. The HPV vaccines were tested on thousands of people and shown to be safe before they were released to the public. These vaccines have been used for years now, and experts say the chance of them causing a serious reaction is very slim. The HPV vaccine does not contain mercury or the preservative thimerosal.

Is there any reason why I shouldn't get this vaccine?

Some people shouldn’t get the vaccine. You definitely don't want to get the HPV vaccine if you've had a serious allergic reaction to it or to any of its components. Let your doctor know if you have any severe allergies to anything, including baker's yeast or latex. Also, talk to your doctor if you have an immune system problem or blood disorder.
If you're pregnant, you'll want to wait to get the HPV vaccine until after your baby is born. In studies, HPV vaccines have not been found to cause any problems in babies whose mothers got the vaccine while pregnant, but pregnant women should not get HPV vaccine, as safety studies are still ongoing.

Could I have side effects from the HPV vaccine?

You could have side effects, but they should be mild. Most people who complain of symptoms after getting the HPV shot have minor issues like pain or swelling at the site of the shot, fever, headache, and nausea.
Sometimes people faint after getting the HPV vaccine or any other vaccination. Sitting down after getting the shot can help prevent you from passing out.

If I get the HPV vaccine, is there a chance I could get HPV?

No. The part of the HPV virus used in both vaccines is inactivated (not live), so it can't cause actual HPV infection.

Hormones, Homones, Hormones....We are talking about COPULINS

These (copulins) hormone-like pseudo-psychological chemicals are emitted by female primates, and allow the female to affect--and even control--the male hypothalamus. Copulins are a mixture of vaginal acids suspended in vaginal fluids and are secreted near the presence of male semen, or become airborne and transmitted to nearby males.
Research since the mid to late 90s has proven that copulins can affect and even control a male's brain. It's long been known that a female's "pheromones" can affect certain male behavior. Pheromones will affect how a male behaves sexually toward the female, but copulins will affect a male's dominant/submissive behavior. Each female's copulins will affect each male's behavior differently and at different levels. A female whose copulins have a strong effect on a particular male may notice the male wanted to please her, offer gifts, and do what she wants. This is not a sexually driven need to please, but an unconscious reflex, and the female may notice there is no limit to what the male will do for her. 

Cumulative effects of copulins on male behavior
Stage 1)
Initial copulin introduction.
The first time a female’s copulins are introduced into a male’s hypothalamus, the effects are usually anticlimactic.  Typical reaction ranges from no effect, a general feeling of well-being, or a state of relaxation.  Because copulins are similar to pheromones, the male may also find the female more attractive, but this is very rare.  The most common reaction is no reaction.  At this stage--which can last just one instance, or many—the male’s polypeptides in the hypothalamus are unaffected, and the receptors in the hypothalamus ignore the copulins.

Stage 2)
Initial copulin reception
At this stage, the male brain begins to receive copulins as input.  As with all these stages, there is no line in the sand for when the first stage ends and the next stage begins.  Some couples never reach this stage.  The male’s receptors have begun to accept copulins as neurotransmitters; that is to say, the brain is no longer ignoring the copulins, and is actually being affected by them.  The male’s polypeptides continue to do their job, however, so the male is now receiving mixed signals.  He may feel slightly confused, but he will still be able to differentiate between his own thoughts and ideas, and those that are foreign.  The female’s input may now sound like better ideas than they did before, but the male does still know these are the female’s ideas and not his own. 
Furthermore, this is the stage where the male begins to develop recurring behaviors during coupling.  Many of the occurrences during this time will become permanent.  For example, if the male says something in particular, it may stick, and he will say this same phrase during subsequent couplings.  I know of one couple where the male always recites the same joke each time they couple, always after about 20 minutes.  This example is somewhat uncommon, for usually the repeated behavior will be something quite ordinary (like saying “I love you.”).  It may seem ordinary, but later becomes unusual in that the behavior repeats at about the same time every coupling.  The behavior is normal—the pattern is odd.  This is a learned behavior, and is caused by when the copulins make their first communication with the hypothalamus.  The hypothalamus remembers the copulins when they become present again, triggering whatever behavior the male exhibited that first time.  Although at this time the male is aware of his behavior, it is a triggered, habitual response, requiring little thought.
The first phases of addiction begin at this stage as well.  As stated before, copulins appear to be highly addictive.  What is not known is whether it is a chemical or psychological addiction, or both.  This addiction starts out as nothing more than a desire to “do that again sometime”, and this desire is strongest immediately after coupling, quickly weakening as time goes by.  We’ll see how this addiction pattern flips in later stages.
In the late phases of this stage, the male may begin exhibiting submissive behavior toward the female, or at least the desire to please.  Many females express their belief that this was caused by the intimacy and eroticism of the situation, and the male’s desire to please in order to garner sexual gratification.  I disagree with this, but at this point have no way to prove or disprove my assertions.

Stage 3)
Copulin ascendancy
This advanced stage occurs with couples who have been together a long time, or when the male’s hypothalamus has been affected by copulins non-stop for an extended time (several weeks or months).  What’s important to remember is that these stages continue to advance, no matter the length of time between couplings.  So, coupling two days in a row will have the same effect as coupling a second time after a year since the first time. 
At this stage, the more aggressive copulins are favored as neurotransmitters over the male’s polypeptides, and the female’s input is primary as the copulins now direct the hypothalamus.  This is not to say that the male is unable to think for himself, but what happens is that all input from the male is regulated at the hypothalamus level.  The hypothalamus acts a relay station, processing two-way information.  First, outside stimulus is first processed at the hypothalamus, which in turn tells the rest of the brain how to respond.  Second, brain activity sends information to the hypothalamus, which in turn tells another part of the brain how to respond.  The male brain can think for itself, unless it is in conflict with input from the female.  The male can think of anything at all at this time, but as soon as input is received from the female, the brain focuses entirely on that input, and all thoughts that are contrary to the female’s input are halted.  For example, the male might be thinking about playing golf on Tuesday, even though he knows the female doesn’t like him to play golf.  If the female stated that he won’t think about golf anymore, and will instead think of her, the male will find himself incapable of thinking of golf while the copulins are present.
At this stage also is when copulins begin to desensitize nerves in the male genitalia.  It’s not understood why copulins don’t have this effect initially, but I’ve speculated that it’s similar to a resistance breakdown.  The more frequent copulins are in contact with nerve endings, the stronger their ability to anesthetize the nerves.  At this stage, the male will experience mild tingling in the genitalia, and sometimes just below the navel.
In this stage or the next, as copulins flood the hypothalamus, the male may also experience hyperactive motor activity; his body may tremble, and his breathing can become short and irregular.  Because the hypothalamus regulates motor activity, I can only assume this is a side effect of the “switch over” from polypeptide to copulin neurotransmission.   

Stage 4)
Copulin dominance
Few couples achieve this level of copulin dominance in the male hypothalamus, and if it does happen, it is because the female has made a conscious effort to keep her copulins active in her mate’s hypothalamus.  In short, this stage is not reached by accident.  At this stage, the hypothalamus completely ignores the male’s polypeptides while copulins are present, even if the female gives no input.  Although the male is not forming too many original thoughts, the brain is still communicating ideas back and forth; however, these ideas are completely regulated by what the male already knows the female expects.  Using the example above, the male might think in the forefront, “What am I going to do on Tuesday?”   Without ever considering golf, the male will answer himself by immediately thinking what the female would want.  What’s important is that the male isn’t making a conscious effort to put himself in his mate’s shoes, but truly thinks he has formulated this idea on his own.  Past communication has already established that the female doesn’t want him to play golf, and so the copulins allow only those thoughts that promote non-golf activities to reach the conscious level.  This level is the classic copulin model that most people think about when it comes to copulins and their effects.  
Also at this level is when copulins can affect the male limbic system where memories reside.  Please remember that nothing is ever completely erased from memory.  In even the most severe amnesia cases, memories have finally been accessed after many years.  What is influenced is the access to the memories, not the memories themselves.  At the female’s input, memories stored in the male’s limbic system can be locked, unable to be accessed by the male.  By the same token, these same memories can be unlocked at a later date.  Also, memories can be planted; that is, something that never happened can be planted as a memory, or an event can be altered.  Females have often reported that when they’ve done anything with their mate’s memories, the male conveys a sensation of ecstasy and profound awe of the female.  I’m not certain why this would happen, but my early premise is that this process (when the copulins manipulate the limbic system) triggers a chemical response similar to those of psycho active drugs. 
The addiction at this level has changed so that the longer the male is without copulins, the more he craves it.  He may crave copulins a day later, but a week later he will think about it often (he will think about coupling, or whatever activity has transmitted copulins in the past), and month later he may become constantly agitated.  The few males I’ve interviewed on this admitted that they had no idea why they had grown increasingly upset, and were unaware that they were addicted to copulins.
Females report that males at this stage, as copulins are transmitting, steadily become “dumber”, repeating themselves or the female’s words, slurring, or mixing words up.  Late in the coupling, females state that the male will typically become incoherent and finally go silent.  Once a hypothalamus is flooded with copulins, the male brain is just sitting on idle, with only the bare minimum of thought process.  In this state, the male is probably not thinking of anything at all, but any input from the female will become the male’s singular focus.  This is the best occasion for accessing the limbic system effectively.
Stage four is also where we begin to see the male’s behavior capable of being modified, even after copulins are no longer present.  The female can plant ideas during coupling (later stages of the session) and the male will act upon them at a later date, completely convinced they are his own idea, even if the male has not had copulins in his system for an extended period.  Just as in hypnosis, trigger words can be used to “trigger” the male to respond in a predetermined fashion.  What is happening here is that the copulin/hypothalamus communication creates new behavior models for the male.  We all have behavior models from birth, and each person’s model is different, of course.  When somebody says “chocolate”, my mouth begins to water, and I may hear my tummy rumble.  My sister, however, may grimace at the word “chocolate”, for she is allergic to it and has learned that chocolate is a bad word.  These are automatic responses, and the male’s response to a trigger word, or modified behavior during a coupling session, is the same—an automatic, unthinking response. 
Whether this behavior modification via copulins is permanent or not is unknown; to date, no male research subject has slipped out of the behavior model set up by the female partner.  Finding enough couples that have reached stage four willing to participate in a long term, controlled study has proven difficult.
The male at this level of copulin dependency may begin to see his female partner as something more than she is.  He may attribute her seeming powers to something supernatural, divine, or having some kind of superiority over himself.  Many males have stated that they learned their mate was superior to them in all ways, even when it was obvious this was not the case.  One well-muscled male, who was considerably larger than his wife, believed she was physically stronger than he was.  Another common testimony by males is that they feel their mate possesses a commanding voice, and can make things happen just by the power of her words.  This sort of admiration the male has for his female partner can border on deification, and one male candidly asserted that his wife could electrocute him if she was so inclined.  There is no way of knowing whether these notions of supernatural power were planted by the females, or were the unwitting fabrications by awestruck males, but after extensive interviews with the female subjects, it’s obvious that the males do at least elevate the female to a level of superiority.
At this stage, we see an increase in the occurrence of hyperactive motor activity when the copulins flood the hypothalamus.  Also, the male will experience more numbness than before, with the genitalia become totally desensitized, and varying levels of numbness from upper thigh to just below the ribcage.  Tingling sensations may travel up the spine, or in the palms and feet.  A few testimonials report the same sensation in the back of the head, or rash-like discoloration on the cheeks, neck, or upper chest.
Late in this stage, the testicles appear to produce less sperm than in earlier stages, and on average, contain more copulins than before.  The copulins have apparently directed the hypothalamus to, in turn, switch off sperm production.  This then permits more volume of copulins to be stored in the testicles.

Stage 5)
Persona Assimilation
In my early studies of couples, I had mapped levels one through four, but in recent years, keeping in touch with many of those couples, I realized there was need for a new level.  The couples in question had all maintained a stage 4 for five years or more.  That is to say that the males had been receiving copulins regularly for many years.  My initial assessment was that the levels of effect reached a plateau, and nothing new developed in male behavior.  Couples had gone four years of almost daily coupling without noticing any positive change in the male’s behavior.  All males exhibited the highest levels of behavior as described in stage 4 above.  In 2002, I started receiving accounts of new behavior, and the next year, these accounts increased.  By early 2004, I was convinced that all of the study couples had risen to a new stage, beyond that described before.  This stage is based on testimony, not on actual controlled study.
Females reported that their male partners were acting on behalf of the female, as though anticipating the female’s needs before those needs were voiced.  “It’s as though he’s reading my mind now,” one female said.  The behavior described verges on how subconscious communication might work, and I received several emails asking if this was some sort of extrasensory perception, or something from the X-Files.  One female asked if she and her husband were now communicating like animals, using some sixth sense.  Let me assure you that there’s a less fantastical explanation.
My presumption is that the male has learned his partner’s traits so well that he “knows” her as well as she knows herself.  This is not caused by anything mysterious, but rather, because the male has made the female his primary focus-- at such a high level, for an unbroken and extended time—he has assimilated the female’s values, character, manners and patterns.  Before, the male performed what the female instructed because he was instructed to do so; however, at this stage, the male performs without instruction because that is what the female would do, or would want done.  Of course, at stage four, the female could instruct the male to always do whatever needs to be done, but if anything new was thrown into the mix, the male would not act upon it.  For example, a female described how she had her husband scrub the inside of the refrigerator every Saturday morning at 8:30 AM, which he faithfully performed for 38 months without fail.  When the refrigerator was replaced, and the old one sat on the curb for pick up, that following Saturday, the male dutifully went out and scrubbed the old refrigerator as always, even though the appliance was now empty and destined for the dump.  In stage five, the male thinks before acting.  What has happened is that we’ve come full circle.
Before a male is introduced to copulins, or is in the early stages, he thinks for himself without input from the female.  Later, the male thinks, but with the female’s input.  Finally, at stage four, the male acts upon the female’s input without thinking.  At stage five, the male is again thinking without input from the female, but what’s different from the first stage is that the male is thinking what the female would think.  The male has effectively become an extension of the female, where the female is the mind, and the male is the body that acts out the mind’s instructions.  To test my theory, I have asked the female couples reporting this stage to try and mix things up for an entire week, to confuse the males with unexpected instructions.  All females reported that when they told their partner to do something uncharacteristic, the male continued to do things “the right way”.  For instance, a female described her partiality for tidiness—a neat freak, in her own words.  She then instructed her male to leave dishes out, toss dirty clothes on the floor, and not pick up anything that had been left out.   The male did as instructed.  “But,” she says, “then the mess started getting on my nerves, and without me saying a word, he immediately cleaned up the mess.  I had planned on cutting your experiment short and having him clean up the mess the next day, but it was as though he picked up on my feelings or thoughts and acted on them.”  Why this persona assimilation occurs could be attributed to repetition, as stated above, but it also could be attributed to something biological, and I hope to begin a controlled study of this late this year.
The last characteristic of this phase is that male ejaculation no longer appears to have any effect on copulin-induced behavior (see the page on How Copulins Work).  In stages one through four, ejaculation eliminates copulins from the hypothalamus, but in stage five, it appears that either the copulins remain in the hypothalamus after ejaculation, or the hypothalamus has been trained to block the influence of testosterone.  Without proper data, I can only guess what is happening at the chemical level. 

How do copulins get from the female to the male? TRANSMISSION OF COPULINS: There are two ways copulins can transmit from female to male: through the air, and by direct genital contact.  copulins and their "support" fluids are produced in the vaginal walls, and a small amount of copulins exists at all times within the vagina, usually near the opening.  When a woman breathes, the movement causes minute amounts of copulins to become airborne.  If she is stationary for an extended period of time, the copulins will eventually exist in the air around her; however, it appears to take some time for copulins to escape clothing fabric and actually become airborne.  Like pheromones, airborne copulins are transmitted from female to male via the male's VNO organ inside the nasal cavity. 1) The first role of copulins is to detect the presence of male semen. When semen comes into contact with the vagina, the copulins trigger the vaginal walls to produce more copulins, and their supporting fluids (see below). 2) Once the copulins detect male semen, the vaginal entry produces lubrication to promote penile entry into the vagina. This is the same fluid that is produced to help intercourse. If intercourse begins immediately, the copulins have no time to take any effect. 3) As the vaginal walls begin to secrete copulins, they are also triggered to expand. This expansion within the vagina creates a vacuum, helping to facilitate the "drawing in" of the penis. 4) If the penis duct remains relatively immobile against any portion of the vaginal wall or entry, the vaginal walls begin to produce copulins at the location of the penis duct (detection of semen). Note that if the penis does not enter the vagina, copulin transmission is relatively slow. Most males experience some loss of feeling as copulins--which are found to have anesthetic qualities--enter the penis. My theory is that this is for those situations where the female needs to get this done without the male knowing it (see my answer to question below). 5) The vagina walls continue to contract and expand in attempts to draw the penis into itself. This is an involuntary muscle response, but also serves to stimulate the penis to become erect. 6) Once the penis is within the vagina, a viscous fluid is produced around the penile shaft. This fluid is non-lubricating, and actually holds the penis in place. Note, this fluid is not copulins themselves, but a fluid that is triggered merely by the presence of something immobile within the vaginal cavity. The penis continues to try to lubricate the vagina with semen, but we'll get to that in a moment.... 7) Now the vagina has the penis where it wants it, and the fun begins. It has long been known that the vagina of primates produces a hormone, progestogen, that shrinks tissue. Researchers postulated that this was for childbirth, to help shrink hemorrhaging infant tissue. However, it has never been explained why progestogen was never present during childbirth, and why it only was found in women who had had intercourse. It is now known that progestogen shrinks the interior walls of the penis. This does not shrink the penis itself, but rather the tissue inside, causing the walls of the penis to pull away from themselves. The penile duct widens and opens. As the penile duct opens, so too the progestogen enters, further opening the penile duct. 8) While the penile duct is being opened, the vaginal walls begin secreting a much thicker fluid, thicker even than the fluid holding the penis in place. The fluid is chemically attracted to semen. In short, the fluid enters the penis, follows the semen down the shaft and directly into the gonads. This process can take as long as fifteen minutes, and requires that the vagina produce up to and exceeding 100 milliliters (a little less than half a cup) of this fluid in order to completely fill the penile shaft and gonads. This is over twenty times as much fluid as is ejaculated by the male during sexual intercourse. 9) Once this unbroken connection between the vaginal wall and gonads is established, the copulins begin traveling down this strand of fluid, to the gonads. Dr. McClintock described this fluid as having the same viscosity as thick honey. My research did not cover this process as it has been well documented by many other researchers. 10) As the copulins travel into the gonads, the vaginal walls begin to contract involuntarily. Normally this would facilitate pushing out the penis, but because the penis has been secured by the sticky fluid from earlier, the pressure causes the copulins and its transmittal fluid to surge into the penis. Fluid and copulin production increases, and measurements of total fluid level is up to a 100 milliliter per hour. 11) Where does all this fluid go? Most of the fluid is absorbed and concentrated in the gonads. The gonads are of a spongy tissue, and when the fluid enters the gonads, it becomes more concentrated. The excess fluid is absorbed by the surrounding tissue. In any case, all copulins suspended within the fluid eventually make it into the bloodstream. However, the more fluid that is held within the gonads, the longer the copulins continue to enter the bloodstream. Two-tenths of a liter of this fluid concentrated within the gonads will send copulins into the bloodstream for 24 to 36 hours. 12) When copulins reach the gonads, they are then either immediately absorbed into the bloodstream, or stored for later. If the gonads become full, they cannot produce their own hormones (testosterone and androstenone) until the fluid has been completely absorbed by the body. 13) Once in the bloodstream, copulins travel throughout the male body. Only until they have reached the hypothalamus do they attach themselves to anything and end their journey. EFFECT OF COPULINS ON THE MALE SUBCONSCIOUS: When copulins are present within the male hypothalamus, many changes occur. 1) copulins "disguise" themselves as the male's polypeptides, which are neurotransmitters--the link of communication between the hypothalamus and the rest of the brain and body. Because copulins are more "aggressive" than polypeptides, they easily supplant the host's neurotransmitters. Moreover, copulins appear to be addictive. 2) By controlling the hypothalamus' communication link with the rest of the body, it also can access the nearby limbic system, which is the brain's center for the retaining of memories. The copulins shut off the link to the limbic system, thereby preventing any current activity from being remembered. The male will have hazy memories of well-being, but cannot remember anything while the limbic system is not communicating. 3) Now here is the process that is still not fully understood: Once the copulins have replaced the polypeptides, the sensory input of the male is controlled. Although the male can receive any normal sensory input (sight, smells, auditory, etc.), the female can vocally change what the male perceives and/or remembers. Research has demonstrated that only the voice of the female who produced the copulins can have any effect. Males shut out all other voices and did not respond to other females' voices in the lab, nor voices from a speaker. How the hypothalamus is caused to filter all but a single voice is what is not understood. During this time, the female can vocally do the following: A) Change, remove, or insert memories. B) Tell the male what he sees, hears, feels, smells, tastes. C) Insert subconscious thoughts that will surface as male "ideas" or behavior later. D) Plant trigger words or actions that can cause thoughts, actions, or sensations in the male at later dates (days, weeks, even months).